The Activation Potential of MOF Is Constrained for Dosage Compensation
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology
Reference36 articles.
1. Activation of transcription through histone H4 acetylation by MOF, an acetyltransferase essential for dosage compensation in Drosophila;Akhtar;Mol. Cell,2000
2. High-resolution ChIP-chip analysis reveals that the Drosophila MSL complex selectively identifies active genes on the male X chromosome;Alekseyenko;Genes Dev.,2006
3. A sequence motif within chromatin entry sites directs MSL establishment on the Drosophila X chromosome;Alekseyenko;Cell,2008
4. Role of the male specific lethal (msl) genes in modifying the effects of sex chromosomal dosage in Drosophila;Bhadra;Genetics,1999
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1. N-Terminus of Drosophila Melanogaster MSL1 Is Critical for Dosage Compensation;2024-09-12
2. N-Terminus of Drosophila Melanogaster MSL1 Is Critical for Dosage Compensation;2024-09-12
3. Mutations of Phosphorylation Sites in MSL1 Protein Do Not Affect Dosage Compensation in Drosophila melanogaster;Doklady Biochemistry and Biophysics;2021-07
4. Sex Chromosome Dosage Compensation;eLS;2019-07-29
5. Progressive dosage compensation during Drosophila embryogenesis is reflected by gene arrangement;EMBO reports;2019-07-09
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