Ras and the FAK Paradox
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology
Reference10 articles.
1. Cool-1 functions as an essential regulatory node for EGFreceptor- and Src-mediated cell growth
2. Reduced cell motility and enhanced focal adhesion contact formation in cells from FAK-deficient mice
3. Activation of Rac1, RhoA, and mitogen-activated protein kinases is required for Ras transformation
4. Selective activation of the JNK signaling cascadeand c-Jun transcriptional activity by the small GTPases Rac and Cdc42Hs
5. Cdc42 regulates anchorage-independent growth and is necessary for Ras transformation
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