Distinct Roles for the XPB/p52 and XPD/p44 Subcomplexes of TFIIH in Damaged DNA Opening during Nucleotide Excision Repair
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology
Reference35 articles.
1. DNA repair. Engagement with transcription;Bootsma;Nature,1993
2. Development of a new easy complementation assay for DNA repair deficient human syndromes using cloned repair genes;Carreau;Carcinogenesis,1995
3. Mutations in the XPD helicase gene result in XP and TTD phenotypes, preventing interaction between XPD and the p44 subunit of TFIIH;Coin;Nat. Genet.,1998
4. Mutations in XPB and XPD helicases found in xeroderma pigmentosum patients impair the transcription function of TFIIH;Coin;EMBO J.,1999
5. Phosphorylation of XPB helicase regulates TFIIH nucleotide excision repair activity;Coin;EMBO J.,2004
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