H2AX Prevents DNA Breaks from Progressing to Chromosome Breaks and Translocations-Reference-Cited by-同舟云学术

H2AX Prevents DNA Breaks from Progressing to Chromosome Breaks and Translocations

Published:2006-01 Issue:2 Volume:21 Page:201-214
ISSN:1097-2765
Container-title:Molecular Cell
language:en
Short-container-title:Molecular Cell

Author:

Franco Sonia,Gostissa Monica,Zha Shan,Lombard David B.,Murphy Michael M.,Zarrin Ali A.,Yan Catherine,Tepsuporn Suprawee,Morales Julio C.,Adams Melissa M.,Lou Zhenkun,Bassing Craig H.,Manis John P.,Chen Junjie,Carpenter Phillip B.,Alt Frederick W.

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology

Reference57 articles.

1. H2AX may function as an anchor to hold broken chromosomal DNA ends in close proximity;Bassing;Cell Cycle,2004

2. Increased ionizing radiation sensitivity and genomic instability in the absence of histone H2AX;Bassing;Proc. Natl. Acad. Sci. USA,2002

3. Histone H2AX: a dosage-dependent suppressor of oncogenic translocations and tumors;Bassing;Cell,2003

4. Dynamic assembly and sustained retention of 53BP1 at the sites of DNA damage are controlled by Mdc1/NFBD1;Bekker-Jensen;J. Cell Biol.,2005

5. Abnormal development of Purkinje cells and lymphocytes in Atm mutant mice;Borghesani;Proc. Natl. Acad. Sci. USA,2000

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