BRCA1 Mutational Complementation Induces Synthetic Viability-Reference-Cited by-同舟云学术

BRCA1 Mutational Complementation Induces Synthetic Viability

Published:2020-06 Issue:5 Volume:78 Page:951-959.e6
ISSN:1097-2765
Container-title:Molecular Cell
language:en
Short-container-title:Molecular Cell

Author:

Nacson Joseph,Di Marcantonio Daniela,Wang Yifan,Bernhardy Andrea J.,Clausen Emma,Hua Xiang,Cai Kathy Q.,Martinez Esteban,Feng Wanjuan,Callén Elsa,Wu Wei,Gupta Gaorav P.,Testa Joseph R.,Nussenzweig André,Sykes Stephen M.,Johnson Neil

Funder

National Cancer Institute

National Heart, Lung, and Blood Institute

U.S. Department of Defense

Susan G. Komen for the Cure

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology

Reference36 articles.

1. Functional and mutational landscapes of BRCA1 for homology-directed repair and therapy resistance;Anantha;eLife,2017

2. Specific killing of BRCA2-deficient tumours with inhibitors of poly(ADP-ribose) polymerase;Bryant;Nature,2005

3. DNA Breaks and End Resection Measured Genome-wide by End Sequencing;Canela;Mol. Cell,2016

4. BRCA1-associated exclusion of 53BP1 from DNA damage sites underlies temporal control of DNA repair;Chapman;J. Cell Sci.,2012

5. ATM loss leads to synthetic lethality in BRCA1 BRCT mutant mice associated with exacerbated defects in homology-directed repair;Chen;Proc. Natl. Acad. Sci. USA,2017

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1. Shieldin and CST co-orchestrate DNA polymerase-dependent tailed-end joining reactions independently of 53BP1-governed repair pathway choice;Nature Structural & Molecular Biology;2024-09-03

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3. Positive and negative regulators of RAD51/DMC1 in homologous recombination and DNA replication;DNA Repair;2024-02

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