Aging Triggers Cytoplasmic Depletion and Nuclear Translocation of the E3 Ligase Mahogunin: A Function for Ubiquitin in Neuronal Survival-Reference-Cited by-同舟云学术

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Aging Triggers Cytoplasmic Depletion and Nuclear Translocation of the E3 Ligase Mahogunin: A Function for Ubiquitin in Neuronal Survival

Published:2017-05 Issue:3 Volume:66 Page:358-372.e7
ISSN:1097-2765
Container-title:Molecular Cell
language:en
Short-container-title:Molecular Cell

Author:

Benvegnù Stefano,Mateo María Inés,Palomer Ernest,Jurado-Arjona Jerónimo,Dotti Carlos G.

Funder

ISCIII

FEDER

Innovation Ingenio-Consolider

Spanish Ministry of Science and Spanish Ministry of Economy and Competitiveness

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology

Reference31 articles.

1. Characterization of mahogunin ring finger-1 expression in mice;Bagher;Pigment Cell Res.,2006

2. Deconstruction for reconstruction: the role of proteolysis in neural plasticity and disease;Bingol;Neuron,2011

3. Induction of tumor cell apoptosis by a proteasome deubiquitinase inhibitor is associated with oxidative stress;Brnjic;Antioxid. Redox Signal.,2014

4. Functional depletion of mahogunin by cytosolically exposed prion protein contributes to neurodegeneration;Chakrabarti;Cell,2009

5. Mahogunin ring finger-1 (MGRN1) suppresses chaperone-associated misfolded protein aggregation and toxicity;Chhangani;Sci. Rep.,2013

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