Mitochondrial bioenergetics and redox dysfunctions in hypercholesterolemia and atherosclerosis-Reference-Cited by-同舟云学术

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Mitochondrial bioenergetics and redox dysfunctions in hypercholesterolemia and atherosclerosis

Published:2020-02 Issue: Volume:71 Page:100840
ISSN:0098-2997
Container-title:Molecular Aspects of Medicine
language:en
Short-container-title:Molecular Aspects of Medicine

Author:

Oliveira Helena C.F.,Vercesi Anibal E.

Funder

Fundação de Amparo a Pesquisa do Estado de São Paulo

CNPq

Publisher

Elsevier BV

Subject

Clinical Biochemistry,Molecular Biology,Molecular Medicine,General Medicine,Biochemistry

Reference149 articles.

1. Cholesterol and 25-hydroxycholesterol inhibit activation of SREBPs by different mechanisms, both involving SCAP and Insigs;Adams;J. Biol. Chem.,2004

2. Hypertriglyceridemia increases mitochondrial resting respiration and susceptibility to permeability transition;Alberici;J. Bioenerg. Biomembr.,2003

3. Mitochondrial ATP-sensitive K(+) channels as redox signals to liver mitochondria in response to hypertriglyceridemia;Alberici;Free Radic. Biol. Med.,2009

4. Hyperlipidemic mice present enhanced catabolism and higher mitochondrial ATP-sensitive K+ channel activity;Alberici;Gastroenterology,2006

5. Mitochondrial energy metabolism and redox responses to hypertriglyceridemia;Alberici;J. Bioenerg. Biomembr.,2011

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