Somatic mutational signatures in polyposis and colorectal cancer-Reference-Cited by-同舟云学术

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Somatic mutational signatures in polyposis and colorectal cancer

Published:2019-10 Issue: Volume:69 Page:62-72
ISSN:0098-2997
Container-title:Molecular Aspects of Medicine
language:en
Short-container-title:Molecular Aspects of Medicine

Author:

Grolleman Judith E.,Díaz-Gay Marcos,Franch-Expósito Sebastià,Castellví-Bel Sergi,de Voer Richarda M.

Funder

Generalitat de Catalunya

Dutch Cancer Society

SCB

Instituto de Salud Carlos III

FEDER

SAF

AGAUR, Generalitat de Catalunya

European Union’s Horizon 2020

Furthermore

European Cooperation in Science and Technology

COST

Publisher

Elsevier BV

Subject

Clinical Biochemistry,Molecular Biology,Molecular Medicine,General Medicine,Biochemistry

Reference103 articles.

1. Inherited variants of MYH associated with somatic G: C-->T: a mutations in colorectal tumors;Al-Tassan;Nat. Genet.,2002

2. Inherited DNA-repair defects in colorectal cancer;AlDubayan;Am. J. Hum. Genet.,2018

3. The repertoire of mutational signatures in human cancer. bioRxiv;Alexandrov,2018

4. Clock-like mutational processes in human somatic cells;Alexandrov;Nat. Genet.,2015

5. A mutational signature in gastric cancer suggests therapeutic strategies;Alexandrov;Nat. Commun.,2015

Cited by 14 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Adenomas from individuals with pathogenic biallelic variants in theMUTYHandNTHL1genes demonstrate base excision repair tumour mutational signature profiles similar to colorectal cancers, expanding potential diagnostic and variant classification applications;2024-08-09

2. Mutational Signatures in Solid Tumors;Advances in Molecular Pathology;2023-11

3. Identifying colorectal cancer caused by biallelic MUTYH pathogenic variants using tumor mutational signatures;Nature Communications;2022-06-06

4. Attribution of Cancer Origins to Endogenous, Exogenous, and Preventable Mutational Processes;Molecular Biology and Evolution;2022-04-26

5. SUITOR: Selecting the number of mutational signatures through cross-validation;PLOS Computational Biology;2022-04-04

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