The causes of cancer revisited: “Mitochondrial malignancy” and ROS-induced oncogenic transformation – Why mitochondria are targets for cancer therapy-Reference-Cited by-同舟云学术

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The causes of cancer revisited: “Mitochondrial malignancy” and ROS-induced oncogenic transformation – Why mitochondria are targets for cancer therapy

Published:2010-04 Issue:2 Volume:31 Page:145-170
ISSN:0098-2997
Container-title:Molecular Aspects of Medicine
language:en
Short-container-title:Molecular Aspects of Medicine

Author:

Ralph Stephen J.,Rodríguez-Enríquez Sara,Neuzil Jiri,Saavedra Emma,Moreno-Sánchez Rafael

Publisher

Elsevier BV

Subject

Clinical Biochemistry,Molecular Biology,Molecular Medicine,General Medicine,Biochemistry

Reference203 articles.

1. Ahn, B.Y., Trinh, D.L., Zajchowski, L.D., Lee, B., Elwi, A.N., Kim, S.W., 2009. Tid1 is a new regulator of p53 mitochondrial translocation and apoptosis in cancer. Oncogene. doi:10.1038/onc.2009.413.

2. The potential of vitamin K3 as an anticancer agent against breast cancer that acts via the mitochondria-related apoptotic pathway;Akiyoshi;Cancer Chemother. Pharmacol.,2009

3. Effects of oxygen on the antioxidant responses of normal and transformed cells;Allen;Exp. Cell Res.,2003

4. Coenzyme Q blocks biochemical but not receptor-mediated apoptosis by increasing mitochondrial antioxidant protection;Alleva;FEBS Lett.,2001

5. Dynamic regulation of PGC-1alpha localization and turnover implicates mitochondrial adaptation in calorie restriction and the stress response;Anderson;Aging Cell,2008

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