PC (16:0/14:0) ameliorates hyperoxia-induced bronchopulmonary dysplasia by upregulating claudin-1 and promoting alveolar type II cell repair

Author:

Hou Weiwei,Yu Boshi,Li Yubai,Yan Xudong,Su Qian,Fang Xiaoyan,Zhou Xiaoguang,Yu Zhangbin

Publisher

Elsevier BV

Reference33 articles.

1. Understanding the impact of infection, inflammation and their persistence in the pathogenesis of bronchopulmonary dysplasia;Balany;Front. Med.,2015

2. Bronchopulmonary dysplasia: 50 years after the original description;Bancalari;Neonatology,2019

3. Type 2 alveolar cells are stem cells in adult lung;Barkauskas;J. Clin. Invest,2013

4. Lung surfactant: Function and composition in the context of development and respiratory physiology;Bernhard;Ann. Anat.,2016

5. Increased palmitoyl-myristoyl-phosphatidylcholine in neonatal rat surfactant is lung specific and correlates with oral myristic acid supply;Bernhard;J. Appl. Physiol.,2011

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