Ku, Artemis, and ataxia-telangiectasia-mutated: Signalling networks in DNA damage-Reference-Cited by-同舟云学术

Ku, Artemis, and ataxia-telangiectasia-mutated: Signalling networks in DNA damage

Published:2008 Issue:4 Volume:40 Page:598-603
ISSN:1357-2725
Container-title:The International Journal of Biochemistry & Cell Biology
language:en
Short-container-title:The International Journal of Biochemistry & Cell Biology

Author:

Morio Tomohiro,Kim Hyeyoung

Publisher

Elsevier BV

Subject

Cell Biology,Biochemistry

Reference20 articles.

1. Enhanced sensitivity and long-term G2 arrest in hydrogen peroxide-treated Ku80-null cells are unrelated to DNA repair defects;Arrington;Free Rad. Biol. Med.,2000

2. Role of DNA-dependent protein kinase in neuronal survival;Chechlacz;J. Neurochem.,2001

3. DNA repair protein Ku80 suppresses chromosomal aberrations and malignant transformation;Difilippantonio;Nature,2000

4. Ku70-deficient embryonic stem cells have increased ionizing radiosensitivity, defective DNA end-binding activity, and inability to support V(D)J recombination;Gu;Proc. Natl. Acad. Sci. U.S.A.,1997

5. Increased oxidative stress in ataxia telangiectasia evidenced by alterations in redox state of brains from Atm-deficient mice;Kamsler;Cancer Res.,2001

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