Activation of mTORC1 disrupted LDL receptor pathway: A potential new mechanism for the progression of non-alcoholic fatty liver disease-Reference-Cited by-同舟云学术

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Activation of mTORC1 disrupted LDL receptor pathway: A potential new mechanism for the progression of non-alcoholic fatty liver disease

Published:2015-04 Issue: Volume:61 Page:8-19
ISSN:1357-2725
Container-title:The International Journal of Biochemistry & Cell Biology
language:en
Short-container-title:The International Journal of Biochemistry & Cell Biology

Author:

Liu Jing,Ma Kun Ling,Zhang Yang,Wu Yu,Hu Ze Bo,Lv Lin Li,Tang Ri Ning,Liu Hong,Ruan Xiong Zhong,Liu Bi Cheng

Funder

Natural Science Foundation of Jiangsu Province

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Cell Biology,Biochemistry

Reference38 articles.

1. Activation of ER stress and mTORC1 suppresses hepatic sortilin-1 levels in obese mice;Ai;J Clin Invest,2012

2. Regulation of hepatic LDL receptors by mTORC1 and PCSK9 in mice;Ai;J Clin Invest,2012

3. Effect of sirolimus on the cholesterol content of aortic arch in ApoE knockout mice;Basso;Transplant Proc,2003

4. Molecular pathways in non-alcoholic fatty liver disease;Berlanga;Clin Exp Gastroenterol,2014

5. A systems biology approach to the pathogenesis of obesity-related nonalcoholic fatty liver disease using reverse phase protein microarrays for multiplexed cell signaling analysis;Calvert;Hepatology,2007

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