Long-term CFTR inhibition modulates 15d-prostaglandin J2 in human pulmonary cells-Reference-Cited by-同舟云学术

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Long-term CFTR inhibition modulates 15d-prostaglandin J2 in human pulmonary cells

Published:2012-06 Issue:6 Volume:44 Page:1009-1018
ISSN:1357-2725
Container-title:The International Journal of Biochemistry & Cell Biology
language:en
Short-container-title:The International Journal of Biochemistry & Cell Biology

Author:

Baudouin-Legros Maryvonne,Colas Julien,Moriceau Sandra,Kelly Mairead,Planelles Gabrielle,Edelman Aleksander,Ollero Mario

Funder

Institut National de la Santé et de la Recherche Médicale

Publisher

Elsevier BV

Subject

Cell Biology,Biochemistry

Reference63 articles.

1. Identification of actin as 15-deoxyΔ12,14 prostaglandin J2 target in neuroblastoma cells: mass spectrometric, computational, and functional approaches to investigate the effect on cytoskeletal derangement;Aldini;Biochemistry,2007

2. Down-regulation of the anti-inflammatory protein annexin A1 in cystic fibrosis knock-out mice and patients;Bensalem;Mol Cell Proteomics,2005

3. Identification of 6-oxo-prostaglandin E1 as a naturally occurring prostanoid generated by rat lung;Berry;Br J Pharmacol,1986

4. Eicosanoid release is increased by membrane destabilisation and CFTR inhibition in Calu-3 cells;Borot;PLoS ONE,2009

5. Inhibition of prostaglandin E2 synthesis by a blocker of epithelial chloride channel;Breuer;Biophys Biochem Res Commun,1989

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1. Cystic Fibrosis Bone Disease: The Interplay between CFTR Dysfunction and Chronic Inflammation;Biomolecules;2023-02-24

2. Pharmacological inhibitors of the cystic fibrosis transmembrane conductance regulator exert off-target effects on epithelial cation channels;Pflügers Archiv - European Journal of Physiology;2022-10-07

3. Abnormal n-6 fatty acid metabolism in cystic fibrosis contributes to pulmonary symptoms;Prostaglandins, Leukotrienes and Essential Fatty Acids;2020-09

4. Impact of secondhand smoke on cystic fibrosis: is there a link to fatty acid metabolism?;Thorax;2019-05-02

5. Overexpression of RANKL in osteoblasts: a possible mechanism of susceptibility to bone disease in cystic fibrosis;The Journal of Pathology;2016-08-22

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