Inhibition of PI3K pathway using BKM120 intensified the chemo-sensitivity of breast cancer cells to arsenic trioxide (ATO)-Reference-Cited by-同舟云学术

Inhibition of PI3K pathway using BKM120 intensified the chemo-sensitivity of breast cancer cells to arsenic trioxide (ATO)

Published:2019-11 Issue: Volume:116 Page:105615
ISSN:1357-2725
Container-title:The International Journal of Biochemistry & Cell Biology
language:en
Short-container-title:The International Journal of Biochemistry & Cell Biology

Author:

Alipour Faranak,Riyahi Niknam,Safaroghli-Azar Ava,Sari Soyar,Zandi Zahra,Bashash Davood^ORCID

Funder

Shahid Beheshti University of Medical Sciences

Publisher

Elsevier BV

Subject

Cell Biology,Biochemistry

Reference56 articles.

1. Mechanism of arsenic trioxide inhibition of cell growth and induction of apoptosis in triple negative MDA-MB-231 breast cancer cell line;Alzuwaidi;FASEB J.,2019

2. Targeting human telomerase RNA component using antisense oligonucleotide induces rapid cell death and increases ATO-induced apoptosis in APL cells;Asghari-Kia;Eur. J. Pharmacol.,2017

3. Inhibition of PI3K signaling pathway enhances the chemosensitivity of APL cells to ATO: proposing novel therapeutic potential for BKM120;Bashash;Eur. J. Pharmacol.,2018

4. Novel pan PI3K inhibitor-induced apoptosis in APL cells correlates with suppression of telomerase: an emerging mechanism of action of BKM120;Bashash;Int. J. Biochem. Cell Biol.,2017

5. BIBR 1532 increases arsenic trioxide-mediated apoptosis in acute promyelocytic leukemia cells: therapeutic potential for APL;Bashash;Anticancer Agents Med. Chem.,2013

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