Molecular mechanisms of muscle atrophy in myotonic dystrophies
Author:
Funder
National Institute of Health
Publisher
Elsevier BV
Subject
Cell Biology,Biochemistry
Reference83 articles.
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2. Expression of a dominant negative CELF protein in vivo leads to altered muscle organization, fiber size and subtype;Berger;PLoS ONE,2011
3. Large CTG repeats trigger p16-dependent premature senescence in myotonic dystrophy type 1 muscle precursor cells;Bigot;American Journal of Pathology,2009
4. Effect of the [CCTG]n repeat expansion on ZNF9 expression in myotonic dystrophy II (DM2);Botta;Biochimica et Biophysica Acta,2006
5. Molecular basis of myotonic dystrophy: expansion of a trinucleotide (CTG) repeats at the 3′ end of a transcript encoding a protein kinase family member;Brook;Cell,1992
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