Down-regulation of NAMPT expression by miR-182 is involved in Tat-induced HIV-1 long terminal repeat (LTR) transactivation-Reference-Cited by-同舟云学术

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Down-regulation of NAMPT expression by miR-182 is involved in Tat-induced HIV-1 long terminal repeat (LTR) transactivation

Published:2013-02 Issue:2 Volume:45 Page:292-298
ISSN:1357-2725
Container-title:The International Journal of Biochemistry & Cell Biology
language:en
Short-container-title:The International Journal of Biochemistry & Cell Biology

Author:

Chen Xin-Yu,Zhang Hong-Sheng,Wu Tong-Chao,Sang Wei-Wei,Ruan Zheng

Funder

National Natural Sciences Foundation of China

Beijing Natural Science Foundation

Beijing Nova Program

National Basic Research Program of China

Publisher

Elsevier BV

Subject

Cell Biology,Biochemistry

Reference48 articles.

1. Preventing HIV-1 Tat-induced neuronal apoptosis using antioxidant enzymes: mechanistic and therapeutic implications;Agrawal;Virology,2007

2. NF-κB activation, dependent on acetylation/deacetylation, contributes to HIF-1 activity and migration of bone metastatic breast carcinoma cells;Bendinelli;Molecular Cancer Research,2009

3. NAD(+), sirtuins, and cardiovascular disease;Borradaile;Current Pharmaceutical Design,2009

4. NAMPT in regulated NAD biosynthesis and its pivotal role in human metabolism;Burgos;Current Medicinal Chemistry,2011

5. SIRT1 is a redox-sensitive deacetylase that is posttranslationally modified by oxidants and carbonyl stress;Caito;FASEB Journal,2010

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