Abnormal cortical lysosomal β-hexosaminidase and β-galactosidase activity at post-synaptic sites during Alzheimer's disease progression-Reference-Cited by-同舟云学术

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Abnormal cortical lysosomal β-hexosaminidase and β-galactosidase activity at post-synaptic sites during Alzheimer's disease progression

Published:2015-01 Issue: Volume:58 Page:62-70
ISSN:1357-2725
Container-title:The International Journal of Biochemistry & Cell Biology
language:en
Short-container-title:The International Journal of Biochemistry & Cell Biology

Author:

Magini Alessandro,Polchi Alice,Tozzi Alessandro,Tancini Brunella,Tantucci Michela,Urbanelli Lorena,Borsello Tiziana,Calabresi Paolo,Emiliani Carla

Funder

Fondazione Cassa di Risparmio di Perugia

Publisher

Elsevier BV

Subject

Cell Biology,Biochemistry

Reference48 articles.

1. Up-regulation of the lysosomal system in experimental models of neuronal injury: implications for Alzheimer's disease;Adamec;Neuroscience,2000

2. Remodeling of sphingolipids by plasma membrane associated enzymes;Aureli;Neurochem Res,2011

3. Cell surface associated glycohydrolases in normal and Gaucher disease fibroblasts;Aureli;J Inherit Metab Dis,2012

4. The glycosphingolipid hydrolases in the central nervous system;Aureli;Mol Neurobiol,2014

5. Thirty years of Alzheimer's disease genetics: the implications of systematic meta-analyses;Bertram;Nat Rev Neurosci,2008

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