CC chemokine receptor-3 (CCR3) antagonists: Improving the selectivity of DPC168 by reducing central ring lipophilicity-Reference-Cited by-同舟云学术

CC chemokine receptor-3 (CCR3) antagonists: Improving the selectivity of DPC168 by reducing central ring lipophilicity

Published:2007-06 Issue:11 Volume:17 Page:2992-2997
ISSN:0960-894X
Container-title:Bioorganic & Medicinal Chemistry Letters
language:en
Short-container-title:Bioorganic & Medicinal Chemistry Letters

Author:

Pruitt James R.,Batt Douglas G.,Wacker Dean A.,Bostrom Lori L.,Booker Shon K.,McLaughlin Erin,Houghton Gregory C.,Varnes Jeffrey G.,Christ David D.,Covington Maryanne,Das Anuk M.,Davies Paul,Graden Danielle,Kariv Ilona,Orlovsky Yevgeniya,Stowell Nicole C.,Vaddi Krishna G.,Wadman Eric A.,Welch Patricia K.,Yeleswaram Swamy,Solomon Kimberly A.,Newton Robert C.,Decicco Carl P.,Carter Percy H.,Ko Soo S.

Publisher

Elsevier BV

Subject

Organic Chemistry,Clinical Biochemistry,Drug Discovery,Pharmaceutical Science,Molecular Biology,Molecular Medicine,Biochemistry

Reference22 articles.

1. Chemokine receptor antagonism as an approach to anti-inflammatory therapy: ‘just right’ or plain wrong?

2. CCR3 is essential for skin eosinophilia and airway hyperresponsiveness in a murine model of allergic skin inflammation

3. The murine CCR3 receptor regulates both the role of eosinophils and mast cells in allergen-induced airway inflammation and hyperresponsiveness

4. Elevated plasma eotaxin levels in patients with acute asthma

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