Gut dysbiosis aggravates cognitive deficits, amyloid pathology and lipid metabolism dysregulation in a transgenic mouse model of Alzheimer's disease-Reference-Cited by-同舟云学术

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Gut dysbiosis aggravates cognitive deficits, amyloid pathology and lipid metabolism dysregulation in a transgenic mouse model of Alzheimer's disease

Published:2023-12 Issue:12 Volume:13 Page:1526-1547
ISSN:2095-1779
Container-title:Journal of Pharmaceutical Analysis
language:en
Short-container-title:Journal of Pharmaceutical Analysis

Author:

Qu Chang,Xu Qing-Qing,Yang Wen,Zhong Mei,Yuan Qiuju,Xian Yan-Fang^ORCID,Lin Zhi-Xiu

Funder

Chinese University of Hong Kong

National Natural Science Foundation of China

Natural Science Foundation of Guangdong Province

Publisher

Elsevier BV

Subject

Analytical Chemistry,Electrochemistry,Spectroscopy,Drug Discovery,Pharmaceutical Science,Pharmacy

Reference45 articles.

1. Alzheimer’s disease;Querfurth;N Engl J. Med.,2010

2. An improved cell-based method for determining the γ-secretase enzyme activity against both Notch and APP substrates;McKee;J. Neurosci. Meth.,2013

3. The role of Cdk5 in Alzheimer’s disease;Liu;Mol. Neurobiol.,2016

4. Role of Cdk5 in amyloid-beta pathology of Alzheimer’s disease;Lu;Curr. Alzheimer Res.,2019

5. Interplay between cyclin-dependent kinase 5 and glycogen synthase kinase 3 beta mediated by neuregulin signaling leads to differential effects on tau phosphorylation and amyloid precursor protein processing;Wen;J. Neurosci.,2008

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