Mitochondria DNA deletions in atherosclerotic hypoperfused brain microvessels as a primary target for the development of Alzheimer's disease-Reference-Cited by-同舟云学术

Mitochondria DNA deletions in atherosclerotic hypoperfused brain microvessels as a primary target for the development of Alzheimer's disease

Published:2005-03 Issue: Volume:229-230 Page:285-292
ISSN:0022-510X
Container-title:Journal of the Neurological Sciences
language:en
Short-container-title:Journal of the Neurological Sciences

Author:

Aliyev Ali,Chen Shu G.,Seyidova Dilara,Smith Mark A.,Perry George,de la Torre Jack,Aliev Gjumrakch

Publisher

Elsevier BV

Subject

Clinical Neurology,Neurology

Reference25 articles.

1. Is non-genetic Alzheimer's disease a vascular disorder with neurodegenerative consequences?;Aliev;J. Alzheimer's Dis.,2002

2. Atherosclerotic lesions and mitochondria DNA deletions in brain microvessels as a central target for the development of human AD and AD-like pathology in a aged YAC AβPP transgenic mice;Aliev;Ann. N. Y. Acad. Sci.,2002

3. Mitochondria and vascular lesions as a central target for the development of Alzheimer's disease and Alzheimer disease-like pathology in transgenic mice;Aliev;Neurol. Res.,2003

4. Vascular hypoperfusion, mitochondria failure and oxidative stress in Alzheimer disease;Aliev;Proc. Indian Natl. Sci. Acad.,2003

5. Neuronal mitochondrial abnormalities in yeast artificial chromosome (YAC) transgenic mice overexpressing amyloid precursor protein (APP);Aliev;Soc. Neurosci.,2000

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