Acute Increases in Plasma Mammalian Target of Rapamycin, Glycogen Synthase Kinase-3β, and Eukaryotic Elongation Factor 2 Phosphorylation After Ketamine Treatment in Three Depressed Patients-Reference-Cited by-同舟云学术

Acute Increases in Plasma Mammalian Target of Rapamycin, Glycogen Synthase Kinase-3β, and Eukaryotic Elongation Factor 2 Phosphorylation After Ketamine Treatment in Three Depressed Patients

Published:2013-06 Issue:12 Volume:73 Page:e35-e36
ISSN:0006-3223
Container-title:Biological Psychiatry
language:en
Short-container-title:Biological Psychiatry

Author:

Yang Chun,Zhou Zhi-qiang,Gao Zhi-qin,Shi Jin-yun,Yang Jian-jun

Publisher

Elsevier BV

Subject

Biological Psychiatry

Reference8 articles.

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3. A randomized trial of an N-methyl-D-aspartate antagonist in treatment-resistant major depression;Zarate;Arch Gen Psychiatry,2006

4. mTOR-dependent synapse formation underlies the rapid antidepressant effects of NMDA antagonists;Li;Science,2010

5. Monitoring ketamine treatment response in a depressed patient via peripheral mammalian target of rapamycin activation;Denk;Am J Psychiatry,2011

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