The A53T Mutation in α-Synuclein Enhances Proinflammatory Activation in Human Microglia Upon Inflammatory Stimulus-Reference-Cited by-同舟云学术

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The A53T Mutation in α-Synuclein Enhances Proinflammatory Activation in Human Microglia Upon Inflammatory Stimulus

Published:2024-07 Issue: Volume: Page:
ISSN:0006-3223
Container-title:Biological Psychiatry
language:en
Short-container-title:Biological Psychiatry

Author:

Krzisch Marine^ORCID,Yuan Bingbing,Chen Wenyu,Osaki Tatsuya,Fu Dongdong,Garrett-Engele Carrie M.,Svoboda Devon S.,Andrykovich Kristin R.,Gallagher Michael D.,Sur Mriganka,Jaenisch Rudolf^ORCID

Funder

Massachusetts Institute of Technology Department of Physics

Publisher

Elsevier BV

Reference45 articles.

1. [11C]-PK11195 PET: Quantification of neuroinflammation and a monitor of anti-inflammatory treatment in Parkinson’s disease?;Bartels;Parkinsonism Relat Disord,2010

2. In vivo imaging of microglial activation with [11C](R)-PK11195 PET in idiopathic Parkinson’s disease;Gerhard;Neurobiol Dis,2006

3. Mutation in the alpha-synuclein gene identified in families with Parkinson’s disease;Polymeropoulos;Science,1997

4. Alpha-synuclein proteins promote pro-inflammatory cascades in microglia: Stronger effects of the A53T mutant;Hoenen;PLoS One,2016

5. Limitations of animal models of Parkinson’s disease;Potashkin;Parkinsons Dis,2010

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