Disengagement of Somatostatin Neurons From Lateral Septum Circuitry by Oxytocin and Vasopressin Restores Social Fear Extinction and Suppresses Aggression Outbursts in a Prader-Willi Syndrome Model-Reference-Cited by-同舟云学术

Disengagement of Somatostatin Neurons From Lateral Septum Circuitry by Oxytocin and Vasopressin Restores Social Fear Extinction and Suppresses Aggression Outbursts in a Prader-Willi Syndrome Model

Published:2023-10 Issue: Volume: Page:
ISSN:0006-3223
Container-title:Biological Psychiatry
language:en
Short-container-title:Biological Psychiatry

Author:

Dromard Yann,Borie Amélie M.,Chakraborty Prabahan,Muscatelli Françoise,Guillon Gilles,Desarménien Michel G.,Jeanneteau Freddy^ORCID

Funder

ANR

FRM

Publisher

Elsevier BV

Subject

Biological Psychiatry

Reference65 articles.

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2. Truncating mutations of MAGEL2 cause Prader-Willi phenotypes and autism;Schaaf;Nat Genet,2013

3. Diagnoses and characteristics of autism spectrum disorders in children with Prader-Willi syndrome;Dykens;J Neurodev Disord,2017

4. Prader–Willi syndrome and Schaaf-Yang syndrome: Neurodevelopmental diseases intersecting at the MAGEL2 gene;Fountain;Dis,2016

5. An early postnatal oxytocin treatment prevents social and learning deficits in adult mice deficient for Magel2, a gene involved in Prader–Willi syndrome and autism;Meziane;Biol Psychiatry,2015

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. MAGEL2 (patho‐)physiology and Schaaf–Yang syndrome;Developmental Medicine & Child Neurology;2024-07

2. Acquiring social safety engages oxytocin neurons in the supraoptic nucleus – role of Magel2 deficiency;2024-02-05