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Transcription factor AP2A affects sFLT1 expression and decidualization in decidual stromal cells: Implications to preeclampsia pathology

  • Published:2020-07 Issue: Volume:21 Page:152-158
  • ISSN:2210-7789
  • Container-title:Pregnancy Hypertension
  • language:en
  • Short-container-title:Pregnancy Hypertension

Author:

Manley Charisma N.,Deepak Venkataraman,Ravikumar Nithin,Smith Alicia K.,Knight Anna K.,Badell Martina L.,Sidell Neil,Rajakumar Augustine

Publisher

Elsevier BV

Subject

Obstetrics and Gynecology,Internal Medicine

Reference33 articles.

1. Gestational hypertension and preeclampsia;ACOG Practice Bulletin No. 202;Obstet. Gynecol.,2019

2. Preeclampsia: pathophysiology, challenges, and perspectives;Rana;Circ. Res.,2019

3. Circulating angiogenic factors and the risk of preeclampsia;Levine;N. Engl. J. Med.,2004

4. Angiogenic factors in preeclampsia and related disorders;Cerdeira;Cold Spring Harb. Perspect. Med.,2012

5. Excess placental soluble fms-like tyrosine kinase 1 (sFlt1) may contribute to endothelial dysfunction, hypertension, and proteinuria in preeclampsia;Maynard;J. Clin. Invest.,2003

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