Genetic polymorphisms of GRIN2A and GRIN2B modify the neurobehavioral effects of low-level lead exposure in children

Author:

Rooney James P.K.,Woods Nancy F.,Martin Michael D.,Woods James S.

Funder

Health Research Board Clinical Fellowship Programme

National Institute of Environmental Health Sciences

Publisher

Elsevier BV

Subject

General Environmental Science,Biochemistry

Reference67 articles.

1. Glutamate receptor, ionotropic, N-methyl D-aspartate 2A (GRIN2A) gene as a positional candidate for attention-deficit/hyperactivity disorder in the 16p13 region;Adams;Mol. Psychiatry,2004

2. Selective blockade of NMDA-activated channel currents may be implicated in learning deficits caused by lead;Alkondon;FEBS Lett.,1990

3. Potential involvement of GRIN2B encoding the NMDA receptor subunit NR2B in the spectrum of Alzheimer's disease;Andreoli;J. Neural Transm.,2014

4. Lead neurotoxicity in children: decomposing the variability in dose-effect relationships;Bellinger;Am. J. Ind. Med.,2007

5. Very low lead exposures and children's neurodevelopment;Bellinger;Curr. Opin. Pediatr.,2008

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