δβ Thalassemia and Hereditary Persistence of Fetal Hemoglobin
Author:
Publisher
Elsevier BV
Subject
Oncology,Hematology
Reference164 articles.
1. Another form of hereditary persistence of fetal hemoglobin (the Atlanta type)?;Altay;Hemoglobin,1977
2. In vitro modulation of γ-gene expression by fetal sheep “switching factor” in two types of (Aγδβ)0 thalassemia;Anagnou;Blood,1998
3. Molecular characterisation of a novel form of (Aγδβ)0 thalassemia deletion with a 3′ breakpoint close to those of HPFH-3 and HPFH-4: Insights into a common regulatory mechanism;Anagnou;Nucl Acids Res,1988
4. Structurally diverse molecular deletions in the β-globin gene cluster exhibit an identical phenotype on interaction with the βs-gene;Anagnou;Blood,1985
5. Sequences located 3′ to the breakpoint of HPFH-3 can modify the developmental expression of the Aγ globin gene;Anagnou;Clin Res,1990
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