Hemochromatosis Protein (HFE) Knockout Mice As a Novel Model of Hemochromatosis: Implications for Study and Management of Iron-Overload Cardiomyopathy

Author:

Zhabyeyev Pavel,Oudit Gavin Y.

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine

Reference28 articles.

1. Balancing acts: molecular control of mammalian iron metabolism;Hentze;Cell,2004

2. Unravelling the molecular basis for cardiac iron metabolism and deficiency in heart failure;Zhabyeyev;Eur Heart J,2016

3. Function of the hemochromatosis protein HFE: lessons from animal models;Pantopoulos;World J Gastroenterol,2008

4. Molecular pathogenesis of hereditary hemochromatosis;Liu;Histol Histopathol,2016

5. HFE and transferrin directly compete for transferrin receptor in solution and at the cell surface;Giannetti;J Biol Chem,2004

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1. Amlodipine rescues advanced iron overload cardiomyopathy in hemojuvelin knockout murine model: Clinical implications;Frontiers in Cardiovascular Medicine;2023-04-21

2. Heme in Cardiovascular Diseases: A Ubiquitous Dangerous Molecule Worthy of Vigilance;Frontiers in Cell and Developmental Biology;2022-01-19

3. Hereditary Hemochromatosis: A Cardiac Perspective;Cureus;2021-11-29

4. Role of iron metabolism in heart failure: From iron deficiency to iron overload;Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease;2019-07

5. TIMP3 deficiency exacerbates iron overload-mediated cardiomyopathy and liver disease;American Journal of Physiology-Heart and Circulatory Physiology;2018-05-01

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