Control of Toll-like Receptor 7 Expression Is Essential to Restrict Autoimmunity and Dendritic Cell Proliferation-Reference-Cited by-同舟云学术

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Control of Toll-like Receptor 7 Expression Is Essential to Restrict Autoimmunity and Dendritic Cell Proliferation

Published:2007-11 Issue:5 Volume:27 Page:801-810
ISSN:1074-7613
Container-title:Immunity
language:en
Short-container-title:Immunity

Author:

Deane Jonathan A.,Pisitkun Prapaporn,Barrett Rebecca S.,Feigenbaum Lionel,Town Terrence,Ward Jerrold M.,Flavell Richard A.,Bolland Silvia

Publisher

Elsevier BV

Subject

Infectious Diseases,Immunology,Immunology and Allergy

Reference41 articles.

1. The Yaa mutation promoting murine lupus causes defective development of marginal zone B cells;Amano;J. Immunol.,2003

2. Selective expansion of a monocyte subset expressing the CD11c dendritic cell marker in the Yaa model of systemic lupus erythematosus;Amano;Arthritis Rheum.,2005

3. Nucleic acids of mammalian origin can act as endogenous ligands for Toll-like receptors and may promote systemic lupus erythematosus;Barrat;J. Exp. Med.,2005

4. Plasmacytoid dendritic cells control TLR7 sensitivity of naive B cells via type I IFN;Bekeredjian-Ding;J. Immunol.,2005

5. Toll-like receptor 7-dependent loss of B cell tolerance in pathogenic autoantibody knockin mice;Berland;Immunity,2006

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