IL-22 Gets to the Stem of Colorectal Cancer
Author:
Publisher
Elsevier BV
Subject
Infectious Diseases,Immunology,Immunology and Allergy
Reference10 articles.
1. Acute Gastrointestinal Infection Induces Long-Lived Microbiota-Specific T Cell Responses
2. DOT1L-Mediated H3K79 Methylation in Chromatin Is Dispensable for Wnt Pathway-Specific and Other Intestinal Epithelial Functions
3. IL-22BP is regulated by the inflammasome and modulates tumorigenesis in the intestine
4. Innate lymphoid cells sustain colon cancer through production of interleukin-22 in a mouse model
5. IL-22+CD4+ T Cells Promote Colorectal Cancer Stemness via STAT3 Transcription Factor Activation and Induction of the Methyltransferase DOT1L
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