Toll-like Receptor 2 Signaling in CD4+ T Lymphocytes Promotes T Helper 17 Responses and Regulates the Pathogenesis of Autoimmune Disease-Reference-Cited by-同舟云学术

Toll-like Receptor 2 Signaling in CD4+ T Lymphocytes Promotes T Helper 17 Responses and Regulates the Pathogenesis of Autoimmune Disease

Published:2010-05 Issue:5 Volume:32 Page:692-702
ISSN:1074-7613
Container-title:Immunity
language:en
Short-container-title:Immunity

Author:

Reynolds Joseph M.,Pappu Bhanu P.,Peng Juan,Martinez Gustavo J.,Zhang Yongliang,Chung Yeonseok,Ma Li,Yang Xuexian O.,Nurieva Roza I.,Tian Qiang,Dong Chen

Publisher

Elsevier BV

Subject

Infectious Diseases,Immunology,Immunology and Allergy

Reference33 articles.

1. The contribution of gammadelta T cells to the pathogenesis of EAE and MS;Blink;Curr. Mol. Med.,2009

2. The development of inflammatory T(H)-17 cells requires interferon-regulatory factor 4;Brüstle;Nat. Immunol.,2007

3. Engagement of TLR2 does not reverse the suppressor function of mouse regulatory T cells, but promotes their survival;Chen;J. Immunol.,2009

4. Critical regulation of early Th17 cell differentiation by interleukin-1 signaling;Chung;Immunity,2009

5. TLR2 engagement on CD8 T cells lowers the threshold for optimal antigen-induced T cell activation;Cottalorda;Eur. J. Immunol.,2006

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