Transcriptional Reprogramming during Effector-to-Memory Transition Renders CD4+ T Cells Permissive for Latent HIV-1 Infection-Reference-Cited by-同舟云学术

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Transcriptional Reprogramming during Effector-to-Memory Transition Renders CD4+ T Cells Permissive for Latent HIV-1 Infection

Published:2017-10 Issue:4 Volume:47 Page:766-775.e3
ISSN:1074-7613
Container-title:Immunity
language:en
Short-container-title:Immunity

Author:

Shan Liang,Deng Kai,Gao Hongbo,Xing Sifei,Capoferri Adam A.,Durand Christine M.,Rabi S. Alireza,Laird Gregory M.,Kim Michelle,Hosmane Nina N.,Yang Hung-Chih,Zhang Hao,Margolick Joseph B.,Li Linghua,Cai Weiping,Ke Ruian,Flavell Richard A.,Siliciano Janet D.,Siliciano Robert F.

Funder

NIH

Beat-HIV

DARE

Johns Hopkins Center for AIDS Research

Howard Hughes Medical Institute

Bill and Melinda Gates Foundation

Mathilde Krim Fellowship, amfAR

National Natural Science Foundation of China

Guangdong Innovative and Entrepreneurial Research Team Program

Joint-Innovation Program in Healthcare for Special Scientific Research Projects of Guangzhou

Publisher

Elsevier BV

Subject

Infectious Diseases,Immunology,Immunology and Allergy

Reference56 articles.

1. Selective escape from CD8+ T-cell responses represents a major driving force of human immunodeficiency virus type 1 (HIV-1) sequence diversity and reveals constraints on HIV-1 evolution;Allen;J. Virol.,2005

2. Administration of vorinostat disrupts HIV-1 latency in patients on antiretroviral therapy;Archin;Nature,2012

3. SAMHD1 restricts HIV-1 infection in resting CD4(+) T cells;Baldauf;Nat. Med.,2012

4. Geno2pheno: Estimating phenotypic drug resistance from HIV-1 genotypes;Beerenwinkel;Nucleic Acids Res.,2003

5. Effect of histone deacetylase inhibitors on HIV production in latently infected, resting CD4(+) T cells from infected individuals receiving effective antiretroviral therapy;Blazkova;J. Infect. Dis.,2012

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