High Glucose Intake Exacerbates Autoimmunity through Reactive-Oxygen-Species-Mediated TGF-β Cytokine Activation-Reference-Cited by-同舟云学术

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High Glucose Intake Exacerbates Autoimmunity through Reactive-Oxygen-Species-Mediated TGF-β Cytokine Activation

Published:2019-10 Issue:4 Volume:51 Page:671-681.e5
ISSN:1074-7613
Container-title:Immunity
language:en
Short-container-title:Immunity

Author:

Zhang Dunfang,Jin Wenwen,Wu Ruiqing,Li Jia,Park Sang-A,Tu Eric,Zanvit Peter,Xu Junji,Liu Ousheng,Cain Alexander,Chen WanJun

Publisher

Elsevier BV

Subject

Infectious Diseases,Immunology,Immunology and Allergy

Reference48 articles.

1. The effect of infections on susceptibility to autoimmune and allergic diseases;Bach;N. Engl. J. Med.,2002

2. Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells;Bettelli;Nature,2006

3. Monocytes from patients with type 1 diabetes spontaneously secrete proinflammatory cytokines inducing Th17 cells;Bradshaw;J. Immunol.,2009

4. Long-term consumption of caffeine-free high sucrose cola beverages aggravates the pathogenesis of EAE in mice;Cao;Cell Discov.,2017

5. Posttranscriptional control of T cell effector function by aerobic glycolysis;Chang;Cell,2013

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