Delayed Sry and Sox9 expression in developing mouse gonads underlies B6-YDOM sex reversal
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Developmental Biology,Molecular Biology
Reference46 articles.
1. DNA sequence analysis of Sry alleles (subgenus Mus) implicates misregulation as the cause of C57BL/6J-Y-pos sex reversal and defines the Sry functional unit;Albrecht;Genetics,1997
2. Evidence that Sry is expressed in pre-Sertoli cells and Sertoli and granulosa cells have a common precursor;Albrecht;Dev. Biol.,2001
3. Defective mesonephric cell migration is associated with abnormal testis cord development in C57BL/6J XY(Mus domesticus) mice;Albrecht;Dev. Biol.,2000
4. Sry expression level and protein isoform differences play a role in abnormal testis development in C57BL/6J mice carrying certain Sry alleles;Albrecht;Genetics,2003
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2. Inefficient Sox9 upregulation and absence of Rspo1 repression lead to sex reversal in the B6.XYTIR mouse gonad;Biology of Reproduction;2024-02-20
3. The −KTS splice variant of WT1 is essential for ovarian determination in mice;Science;2023-11-03
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