DC-SCRIPT deficiency delays mouse mammary gland development and branching morphogenesis-Reference-Cited by-同舟云学术

DC-SCRIPT deficiency delays mouse mammary gland development and branching morphogenesis

Published:2019-11 Issue:1 Volume:455 Page:42-50
ISSN:0012-1606
Container-title:Developmental Biology
language:en
Short-container-title:Developmental Biology

Author:

Tang Chunling,van den Bijgaart Renske J.E.,Looman Maaike W.G.,Tel-Karthaus Nina,de Graaf Annemarie M.A.,Gilfillan Susan,Colonna Marco,Ansems Marleen,Adema Gosse J.

Funder

KWF Kankerbestrijding

Nederlandse Organisatie voor Wetenschappelijk Onderzoek

China Scholarship Council

Publisher

Elsevier BV

Subject

Cell Biology,Developmental Biology,Molecular Biology

Reference44 articles.

1. Nuclear receptor modulation and prognostic significance in primary breast cancer;Ansems;J. Natl. Cancer Inst.,2010

2. Crosstalk and DC-SCRIPT: expanding nuclear receptor modulation;Ansems;Biochim. Biophys. Acta,2010

3. AR and VDR regulator lost upon transformation of prostate epithelial cells;Ansems;Prostate,2012

4. DC-SCRIPT is a novel regulator of the tumor suppressor gene CDKN2B and induces cell cycle arrest in ERalpha-positive breast cancer cells;Ansems;Breast Canc. Res. Treat.,2015

5. Control of mammary stem cell function by steroid hormone signalling;Asselin-Labat;Nature,2010

Cited by 5 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Organoid Cultures for the Study of Mammary Biology and Breast Cancer: The Promise and Challenges;Cold Spring Harbor Perspectives in Medicine;2023-12-18

2. Cancer invasion and metastasis: Insights from murine pubertal mammary gland morphogenesis;Biochimica et Biophysica Acta (BBA) - General Subjects;2023-08

3. Type 1 conventional dendritic cell fate and function are controlled by DC-SCRIPT;Science Immunology;2021-04-02

4. Mammary Organoids and 3D Cell Cultures: Old Dogs with New Tricks;Journal of Mammary Gland Biology and Neoplasia;2020-11-18

5. DC-SCRIPT affects mammary organoids branching morphogenesis by modulating the FGFR1-pERK signaling axis;Developmental Biology;2020-07