Myosin heavy chain mutations that cause Freeman-Sheldon syndrome lead to muscle structural and functional defects in Drosophila-Reference-Cited by-同舟云学术

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Myosin heavy chain mutations that cause Freeman-Sheldon syndrome lead to muscle structural and functional defects in Drosophila

Published:2019-05 Issue:2 Volume:449 Page:90-98
ISSN:0012-1606
Container-title:Developmental Biology
language:en
Short-container-title:Developmental Biology

Author:

Das Shreyasi,Kumar Pankaj,Verma Aakanksha,Maiti Tushar K.,Mathew Sam J.^ORCID

Funder

Wellcome Trust/DBT India Alliance through an intermediate fellowship

CSIR

Stocks obtained from the Bloomington Drosophila Stock Center

Publisher

Elsevier BV

Subject

Cell Biology,Developmental Biology,Molecular Biology

Reference28 articles.

1. Assaying locomotor, learning, and memory deficits in Drosophila models of neurodegeneration;Ali;J. Visualized Exp. JoVE,2011

2. Muscle Lim Protein and myosin binding protein C form a complex regulating muscle differentiation;Arvanitis;Biochim. Biophys. Acta (BBA) Mol. Cell Res.,2017

3. SALS, a WH2-domain-containing protein, promotes sarcomeric actin filament elongation from pointed ends during Drosophila muscle growth;Bai;Dev. Cell,2007

4. Arthrogryposis: a review and update;Bamshad;J. Bone Joint Surg. Am.,2009

5. Genotype-phenotype relationships in Freeman–Sheldon syndrome;Beck;Am. J. Med. Genet. Part A,2014

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