Multiple cardiovascular defects caused by the absence of alternatively spliced segments of fibronectin
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Developmental Biology,Molecular Biology
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4. Astrof, S., Kirby, A., Lindblad-Toh, K., Daly, M.J., Hynes, R.O., in press. Heart development in fibronectin-null mice is goverened by a genetic modifier on chromosome four. Mech. Dev.
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