Dicer inactivation in osteoprogenitor cells compromises fetal survival and bone formation, while excision in differentiated osteoblasts increases bone mass in the adult mouse-Reference-Cited by-同舟云学术

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Dicer inactivation in osteoprogenitor cells compromises fetal survival and bone formation, while excision in differentiated osteoblasts increases bone mass in the adult mouse

Published:2010-04 Issue:1 Volume:340 Page:10-21
ISSN:0012-1606
Container-title:Developmental Biology
language:en
Short-container-title:Developmental Biology

Author:

Gaur Tripti,Hussain Sadiq,Mudhasani Rajini,Parulkar Isha,Colby Jennifer L.,Frederick Dana,Kream Barbara E.,van Wijnen Andre J.,Stein Janet L.,Stein Gary S.,Jones Stephen N.,Lian Jane B.

Funder

NIH

American Heart Award

NIH-T32CA130807

Publisher

Elsevier BV

Subject

Cell Biology,Developmental Biology,Molecular Biology

Reference63 articles.

1. Matricellular proteins: extracellular modulators of bone development, remodeling, and regeneration;Alford;Bone,2006

2. Biological basis of bone formation, remodeling, and repair-part I: biochemical signaling molecules;Allori;Tissue Eng. Part B Rev.,2008

3. The miRNA-processing enzyme dicer is essential for the morphogenesis and maintenance of hair follicles;Andl;Curr. Biol.,2006

4. High bone mass in mice expressing a mutant LRP5 gene;Babij;J. Bone Miner. Res.,2003

5. Dicer is essential for mouse development;Bernstein;Nat. Genet.,2003

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