Effect of aspirin on the fibrinolytic response in perfused rat hindquarters
Author:
Publisher
Elsevier BV
Subject
Pharmacology
Reference16 articles.
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2. Low dose aspirin does not prevent fibrinolytic response to venous occlusion;De Gaetano;Biochem. Pharmacol.,1986
3. Perfused rat hindlegs. A model to study plasminogen activator release;Emeis;Thromb. Res.,1983
4. Aspirin-induced decline in prostacyclin production in patients with coronary artery disease is due to decreased endoperoxide shift. Analysis of the effects of a combination of aspirin and n-3 fatty acids on the eicosanoid profile;Force;Circulation,1991
5. Enhancement by interleukin 1 (IL-1) of plasminogen activator inhibitor (PAI) activity in cultured human endothelial cells;Gramse;Biochem. Biophys. Res. Commun.,1986
Cited by 4 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Alternative Mechanism of Aspirin in Anti-Thrombotic Therapy: Inhibition of Thrombin Activatable Fibrinolysis Inhibitor;Bulletin of the Korean Chemical Society;2012-09-20
2. Aspirin and the fibrinolytic response;Thrombosis Research;2003-06
3. Acute release of t-pa in rats and mice is inhibited by endotoxin and dexamethasone but is not affected by retinoic acid;Fibrinolysis;1994-09
4. Prostacyclin is required for t-PA release after venous occlusion;American Journal of Physiology-Heart and Circulatory Physiology;1994-02-01
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