Both cell surface and internalized β-adrenoceptors are reduced in the failing myocardium
Author:
Publisher
Elsevier BV
Subject
Pharmacology
Reference29 articles.
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2. Decreased catecholamine sensitivity and β-adrenergic-receptor density in failing human hearts;Bristow;N. Engl. J. Med.,1982
3. β1- and β2-adrenergic-receptor subpopulations in nonfailing and failing human myocardium: coupling of both receptor subtypes to muscle contraction and selective β1-receptor down-regulation in heart failure;Bristow;Circ. Res.,1986
4. Dysfunction of the β- and α-adrenergic systems in a model of congestive heart failure. The pacing-overdrive dog;Calderone;Circ. Res.,1991
5. Evidence for internalization of the recognition site of β-adrenergic receptors during receptor subsensitivity induced by (−)-isoproterenol;Chuang,1979
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2. Modification of beta-adrenoceptor signal transduction pathway by genetic manipulation and heart failure;Molecular and Cellular Biochemistry;2000
3. ACE inhibition improves cardiac NE uptake and attenuates sympathetic nerve terminal abnormalities in heart failure;American Journal of Physiology-Heart and Circulatory Physiology;1999-10-01
4. Effect of Digoxin on Ventricular Remodeling and Responsiveness of β-Adrenoceptors in Chronic Volume Overload;Journal of Cardiovascular Pharmacology and Therapeutics;1998-12
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