Chronic exposure of NG108-15 cells to inhibitory acting drugs reduces stimulatory prostaglandin E1 receptor number
Author:
Publisher
Elsevier BV
Subject
Pharmacology
Reference31 articles.
1. Agonist regulation of cellular levels of the stimulatory guanine nucleotide-binding protein, Gs, in wild type and transfected neuroblastoma-glioma hybrid NG 108-15 cells;Adie;Biochem. Soc. Trans.,1993
2. Concurrent down-regulation of IP prostanoid receptors and the α-subunit of the stimulatory guanine-nucleotide-binding protein (Gs) during prolonged exposure of neuroblastoma × glioma cells to prostanoid agonists;Adie;Biochem. J.,1992
3. Coupling of prostaglandin E1 receptors to the stimulatory GTP-binding protein Gs is enhanced in neuroblastoma × glioma (NG 108-15) hybrid cells chronically exposed to an opioid;Ammer;Mol. Pharmacol.,1993
4. Chronic activation of inhibitory δ-opioid receptors cross-regulates the stimulatory adenylate cyclase-coupled prostaglandin E1 receptor system in neuroblastoma × glioma (NG 108-15) hybrid cells;Ammer;J. Neurochem.,1995
5. Opiate-dependent changes in the sensitivity of adenylate cyclase to stimulatory agonists and 5′-guanylylimidodiphosphate are independent of G protein abundance and eukaryotic ADP-ribosyltransferase activity in NG 108-15 cells;Boyd;J. Neurochem.,1992
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