Rapid increase in inducible nitric oxide synthase gene expression in the heart during endotoxemia-Reference-Cited by-同舟云学术

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Rapid increase in inducible nitric oxide synthase gene expression in the heart during endotoxemia

Published:1996-05 Issue:1-2 Volume:303 Page:141-144
ISSN:0014-2999
Container-title:European Journal of Pharmacology
language:en
Short-container-title:European Journal of Pharmacology

Author:

Bateson Alan N.,Jakiwczyk Olga M.,Schulz Richard

Publisher

Elsevier BV

Subject

Pharmacology

Reference15 articles.

1. Myocardial function in sepsis and endotoxin shock;Abel;Am. J. Physiol.,1989

2. Cytokine-inducible nitric oxide synthase (iNOS) expression in cardiac myocytes;Balligand;J. Biol. Chem.,1994

3. Induction of NO synthase in rat cardiac microvascular endothelial cells by IL-1 β and IFN-γ;Balligand;Am. J. Physiol.,1995

4. Nitric oxide production within cardiac myocytes reduces their contractility in endotoxemia;Brady;Am. J. Physiol.,1992

5. Nitric oxide attenuates cardiac myocyte contraction;Brady;Am. J. Physiol.,1993

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3. MCP-1-induced protein attenuates endotoxin-induced myocardial dysfunction by suppressing cardiac NF-кB activation via inhibition of IкB kinase activation;Journal of Molecular and Cellular Cardiology;2011-08

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