Hypoxic coronary vasodilatation and cGMP overproduction are blocked by a nitric oxide synthase inhibitor, but not by a guanylyl cyclase ANF receptor antagonist

Author:

Park Kwan Ha,levi Roberto

Publisher

Elsevier BV

Subject

Pharmacology

Reference15 articles.

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2. Release of endothelium-derived relaxing factor(s) by physiocochemical stimuli;Bassenge,1988

3. Effects of alpha-human atrial natriuretic peptide in guinea-pig isolated heart;Di Nardo;Int. J. Cardiol.,1993

4. Signal transduction mechanisms involving nitric oxide;Ignarro;Biochem. Pharmacol.,1991

5. Inhibition by HS-142-1, a novel nonpeptide atrial natriuretic peptide antagonist of microbial origin, of atrial natriuretic peppeptide antagonist of of isolated rabbit aorta through the blockade of guanylyl cyclase-linked receptors;Imura;Nol. Pharmacol.,1992

Cited by 4 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献

1. Increased release of nitric oxide in ischemic hearts after exercise in patients with effort angina;Journal of the American College of Cardiology;1998-07

2. Role of guanylyl cyclase receptors for CNP in salt secretion by shark rectal gland;American Journal of Physiology-Regulatory, Integrative and Comparative Physiology;1997-10-01

3. Acute hypoxia upregulates NOS gene expression in rats;American Journal of Physiology-Regulatory, Integrative and Comparative Physiology;1997-09-01

4. Neutral endopeptidase 24.11 in neutrophils modulates protective effects of natriuretic peptides against neutrophils-induced endothelial cytotoxity.;Journal of Clinical Investigation;1996-05-15

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