An L-arginine-derived factor mediates endotoxin-induced vascular hyposensitivity to calcium

Author:

Gray Gillian A.,Julou-Schaeffer Géraldine,Oury Kleber,Fleming Ingrid,Parratt James R.,Stoclet Jean-Claude

Publisher

Elsevier BV

Subject

Pharmacology

Reference10 articles.

1. Nitric oxide activates guanylate cyclase and increases guanosine 3'5' cyclic monophosphate levels in various tissue preparations;Arnold,1977

2. Bigaud M., G. Julou-Schaeffer, J.C. Stoclet and J.R. Parratt, Endotoxin-induced impairment of vascular smooth muscle contraction elicited through different mechanisms, European J. Pharmacol. (in press).

3. L-NG-nitro-arginine and its methyl ester are potent inhibitors of non-adrenergic, noncholinergic transmission in the rat anococcygeus;Hobbs;Br. J. Pharmacol.,1990

4. Julou-Schaeffer G., G.A. Gray, I. Fleming, C. Schott, J.R. Parratt and J.C. Stoclet, Loss of vascular responsiveness induced by endotoxin involves the L-arginine pathway, Am. J. Physiol. (in press).

5. Cyclic GMP and mechanisms of vasodilatation;Lincoln;Pharmacol. Ther.,1989

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