Na+,K+-ATPase activity is selectively increased in thalamus in thiamine deficiency prior to the appearance of neurological symptoms
Author:
Publisher
Elsevier BV
Subject
Pharmacology
Reference18 articles.
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3. Cytoarchitectural relationships between [3H]ouabain binding and mRNA for isoforms of the sodium pump catalytic subunit in rat brain;Brines;Mol. Brain Res.,1991
4. Cerebral thiamine-dependent enzyme changes in experimental Wernicke's encephalopathy;Butterworth;Metab. Brain Dis.,1986
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1. Effects of gestational thiamine-deprivation and/or exposure to ethanol on crucial offspring rat brain enzyme activities;The Journal of Maternal-Fetal & Neonatal Medicine;2019-09-25
2. Experimentally-induced Wernicke’s encephalopathy modifies crucial rat brain parameters: the importance of Na+,K+-ATPase and a potentially neuroprotective role for antioxidant supplementation;Metabolic Brain Disease;2013-03-12
3. Gene expression changes in thalamus and inferior colliculus associated with inflammation, cellular stress, metabolism and structural damage in thiamine deficiency;European Journal of Neuroscience;2006-03
4. Vesicular dysfunction during experimental thiamine deficiency is indicated by alterations in dopamine metabolism;European Journal of Pharmacology;1996-12
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