Chronic treatment with SCH 23390, a selective dopamine D-1 receptor antagonist, decreases dopamine metabolism in rat caudate nucleus-Reference-Cited by-同舟云学术

Chronic treatment with SCH 23390, a selective dopamine D-1 receptor antagonist, decreases dopamine metabolism in rat caudate nucleus

Published:1988-10 Issue:3 Volume:155 Page:313-316
ISSN:0014-2999
Container-title:European Journal of Pharmacology
language:en
Short-container-title:European Journal of Pharmacology

Author:

Koulu Markku,Lappalainen Jaakko,Pesonen Ullamari,Hietala Jarmo,Syvälahti Erkka

Publisher

Elsevier BV

Subject

Pharmacology

Reference10 articles.

1. The D-1 dopamine receptor antagonist SCH 23390 also interacts potently with brain serotonin (5HT2) receptors;Bischoff;European J. Pharmacol.,1986

2. Selective D-1 receptor increase following chronic treatment with SCH 23390;Creese;European J. Pharmacol.,1985

3. Chronic haloperidol inactivates brain noradrenergic neurons;Dinan;Brain Res.,1985

4. Effects of chronic SCH23390 treatment on the biochemical and behavioral properties of D1 and D2 dopamine receptors: potentiated behavioral responses to a D2 dopamine agonist after selective D1 dopamine receptor upregulation;Hess;J. Pharmacol. Exp. Ther.,1986

5. In vivo receptor binding, neurochemical and functional studies with the dopamine D-1 receptor antagonist SCH 23390;Hjorth;J. Neural Transm.,1988

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2. Enhancement of excessive lever-pressing after post-training signal attenuation in rats by repeated administration of the D-1 antagonist SCH 23390 or the D-2 agonist quinpirole, but not the D-1 agonist SKF 38393 or the D-2 antagonist haloperidol;BEHAV NEUROSCI;2001

3. Tyrosine hydroxylase, aromatic l-amino acid decarboxylase and dopamine metabolism after chronic treatment with dopaminergic drugs;Brain Research;1999-06

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