The phospholipase A2 superfamily as a central hub of bioactive lipids and beyond

Author:

Murakami Makoto

Funder

Japan Agency for Medical Research and Development

Japan Society for the Promotion of Science

Publisher

Elsevier BV

Subject

Pharmacology (medical),Pharmacology

Reference369 articles.

1. Progesterone-induced acrosome exocytosis requires sequential involvement of calcium-independent phospholipase A2β (iPLA2β) and group X secreted phospholipase A2 (sPLA2);Abi Nahed;Journal of Biological Chemistry,2016

2. Diabetes and hypercholesterolemia increase blood-brain barrier permeability and brain amyloid deposition: beneficial effects of the LpPLA2 inhibitor darapladib;Acharya;Journal of Alzheimer’s Disease,2013

3. Inherited human cPLA2α deficiency is associated with impaired eicosanoid biosynthesis, small intestinal ulceration, and platelet dysfunction;Adler;Journal of Clinical Investigation,2008

4. Brain-specific deletion of neuropathy target esterase/swisscheese results in neurodegeneration;Akassoglou;Proceedings of the National Academy of Sciences of the United States of America,2004

5. S-adenosylmethionine (SAMe) therapy in liver disease: A review of current evidence and clinical utility;Anstee;Journal of Hepatology,2012

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