Posttranslational modifications of histone deacetylases: Implications for cardiovascular diseases-Reference-Cited by-同舟云学术

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Posttranslational modifications of histone deacetylases: Implications for cardiovascular diseases

Published:2014-08 Issue:2 Volume:143 Page:168-180
ISSN:0163-7258
Container-title:Pharmacology & Therapeutics
language:en
Short-container-title:Pharmacology & Therapeutics

Author:

Eom Gwang Hyeon,Kook Hyun

Funder

National Research Foundation of Korea grant funded by the Korean government (MEST

National Research Foundation of Korea grant (MRC

Korea government (MSIP)

Korean Health Technology R&D Project, Ministry of Health & Welfare, Republic of Korea

Publisher

Elsevier BV

Subject

Pharmacology (medical),Pharmacology

Reference157 articles.

1. A redox-dependent pathway for regulating class II HDACs and cardiac hypertrophy;Ago;Cell,2008

2. Trichostatin A, an HDAC class I/II inhibitor, promotes pi-induced vascular calcification via up-regulation of the expression of alkaline phosphatase;Azechi;J Atheroscler Thromb,2013

3. High-resolution profiling of histone methylations in the human genome;Barski;Cell,2007

4. Tumor necrosis factor and interleukin-1 lead to phosphorylation and loss of I kappa B alpha: a mechanism for NF-kappa B activation;Beg;Mol Cell Biol,1993

5. Molecular distinction between physiological and pathological cardiac hypertrophy: experimental findings and therapeutic strategies;Bernardo;Pharmacol Ther,2010

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