Generation of human induced pluripotent stem cell lines from sporadic, sporadic frontotemporal dementia, familial SOD1, and familial C9orf72 amyotrophic lateral sclerosis (ALS) patients
Author:
Funder
Motor Neurone Disease Research Australia
Bonneville Power Administration
University of Queensland
University of Western Australia
Australian Government
FightMND
Bioplatforms Australia
National Health and Medical Research Council
Publisher
Elsevier BV
Reference7 articles.
1. Expanded ggggcc hexanucleotide repeat in noncoding region of c9orf72 causes chromosome 9p-linked ftd and als;DeJesus-Hernandez;Neuron,2011
2. Human ipsc-derived motoneurons harbouring tardbp or c9orf72 als mutations are dysfunctional despite maintaining viability;Devlin;Nat. Commun.,2015
3. Modeling sporadic als in ipsc-derived motor neurons identifies a potential therapeutic agent;Fujimori;Nat. Med.,2018
4. Altered tdp-43 structure and function: Key insights into aberrant rna, mitochondrial, and cellular and systemic metabolism in amyotrophic lateral sclerosis;Jiang;Metabolites,2022
5. Nutritional and metabolic factors in amyotrophic lateral sclerosis;Ludolph;Nat. Rev. Neurol.,2023
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