S-Adenosyl-l-methionine and alcoholic liver disease in animal models

Author:

Lieber Charles S

Publisher

Elsevier BV

Subject

Behavioral Neuroscience,Neurology,Toxicology,General Medicine,Biochemistry,Health (social science)

Reference35 articles.

1. Polyenylphosphatidylcholine opposes the increase of cytochrome P-4502E1 by ethanol and corrects its iron-induced decrease;Aleynik;Alcohol Clin Exp Res,1999

2. Polyenylphosphatidylcholine (PPC) corrects the alcohol-induced hepatic oxidative stress by restoring S-adenosyl-l-methionine (SAMe);Aleynik;Alcohol Clin Exp Res,2001

3. Decreased cytochrome oxidase activity in hepatic mitochondria after chronic ethanol consumption and the possible role of decreased cytochrome aa3 content and changes in phospholipids;Arai;Biochim Biophys Acta,1984

4. Pitfalls and problems in studies on the methylation of phosphatidylethanolamine;Audubert;J Biol Chem,1983

5. Adaptive increase in betaine-homocysteine methyltransferase activity maintains hepatic S-adenosyl-l-methionine levels in ethanol-treated rats;Barak;IRCS Med Sci Biochem,1984

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