Aflatoxin B1 exposure deteriorates immune abnormalities in a BTBR T+ Itpr3tf/J mouse model of autism by increasing inflammatory mediators' production in CD19-expressing cells-Reference-Cited by-同舟云学术

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Aflatoxin B1 exposure deteriorates immune abnormalities in a BTBR T+ Itpr3tf/J mouse model of autism by increasing inflammatory mediators' production in CD19-expressing cells

Published:2024-06 Issue: Volume:391 Page:578365
ISSN:0165-5728
Container-title:Journal of Neuroimmunology
language:en
Short-container-title:Journal of Neuroimmunology

Author:

Almanaa Taghreed N.,Alwetaid Mohammad Y.,Bakheet Saleh A.,Attia Sabry M.,Ansari Mushtaq A.,Nadeem Ahmed,Ahmad Sheikh F.

Funder

King Saud University

Publisher

Elsevier BV

Reference114 articles.

1. Intracellular pathogen infections and immune response in autism;Abib;Neuroimmunomodulation,2018

2. Not(ch) just development: notch signalling in the adult brain;Ables;Nat. Rev. Neurosci.,2011

3. The Stat3 inhibitor, S3I-201, downregulates lymphocyte activation markers, chemokine receptors, and inflammatory cytokines in the BTBR T+ Itpr3tf/J mouse model of autism;Ahmad;Brain Res. Bull.,2019

4. Methylmercury chloride exposure aggravates proinflammatory mediators and Notch-1 signaling in CD14(+) and CD40(+) cells and is associated with imbalance of neuroimmune function in BTBR T(+) Itpr3tf/J mice;Ahmad;Neurotoxicology,2021

5. Cadmium exposure is associated with behavioral deficits and Neuroimmune dysfunction in BTBR T+ Itpr3tf/J mice;Alanazi;Int. J. Mol. Sci.,2023

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